INT241614

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Context Info
Confidence 0.06
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.37

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (CCNA2) mitosis (CCNA2) extracellular region (PRB3)
nucleus (CCNA2) cell division (CCNA2) biological_process (PRB3)
CCNA2 (Homo sapiens)
PRB3 (Homo sapiens)
Pain Link Frequency Relevance Heat
cocaine 292 99.32 Very High Very High Very High
Central nervous system 10 5.00 Very Low Very Low Very Low
monoamine 3 5.00 Very Low Very Low Very Low
cerebral cortex 2 5.00 Very Low Very Low Very Low
Neurotransmitter 2 5.00 Very Low Very Low Very Low
Neurobehavioral 2 5.00 Very Low Very Low Very Low
sodium channel 1 5.00 Very Low Very Low Very Low
local anesthetic 1 5.00 Very Low Very Low Very Low
gABA 1 5.00 Very Low Very Low Very Low
Dopamine 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Intellectual Impairment 1 40.12 Quite Low
Cocaine Dependence 7 31.92 Quite Low
Congenital Anomalies 5 30.00 Quite Low
Stress 27 25.00 Low Low
Toxicity 4 25.00 Low Low
Apoptosis 3 25.00 Low Low
Reprotox - General 1 1 25.00 Low Low
Pre-term Labor 1 25.00 Low Low
Neurodegenerative Disease 22 5.00 Very Low Very Low Very Low
Death 8 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Next, the researchers showed that cocaine-treated AF5 cells made much less cyclin A2, a protein that controls the G1-to-S transition, than untreated cells.
cyclin A2 Regulation (controls) of G1 associated with cocaine
1) Confidence 0.06 Published 2008 Journal PLoS Medicine Section Abstract Doc Link PMC2504032 Disease Relevance 0 Pain Relevance 0.37

General Comments

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