INT242483

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Context Info
Confidence 0.17
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.76
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Ciapin1, Bax) nucleus (Ciapin1, Bax) mitochondrion (Bax)
endoplasmic reticulum (Bax) intracellular (Bax) lipid binding (Bax)
Ciapin1 (Mus musculus)
Bax (Mus musculus)
Pain Link Frequency Relevance Heat
Palliative 1 5.00 Very Low Very Low Very Low
Cytokine 1 5.00 Very Low Very Low Very Low
adenosine 1 5.00 Very Low Very Low Very Low
Potency 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 37 99.10 Very High Very High Very High
Hepatocellular Cancer 78 95.48 Very High Very High Very High
Cancer 57 86.56 High High
Infection 24 84.92 Quite High
Death 7 47.24 Quite Low
Targeted Disruption 7 5.00 Very Low Very Low Very Low
Hepatitis 6 5.00 Very Low Very Low Very Low
Hematological Disease 4 5.00 Very Low Very Low Very Low
Cirrhosis 4 5.00 Very Low Very Low Very Low
Adenovirus Infection 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Furthermore, analysis of Bcl-2 family mediators of apoptosis, such as Bcl-2, Bax and Bcl-XL, showed that depletion of CIAPIN1 could downregulate Bcl-2, Bcl-XL and upregulate Bax in AdSiCIAPIN1-transduced cells, suggesting that the apoptotic effect of CIAPIN1 could be partly mediated by the Bcl-2 family (Figure 4D).


Negative_regulation (depletion) of CIAPIN1 Negative_regulation (downregulate) of Positive_regulation (upregulate) of Bax associated with apoptosis
1) Confidence 0.17 Published 2008 Journal Carcinogenesis Section Body Doc Link PMC2516489 Disease Relevance 1.76 Pain Relevance 0

General Comments

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