INT242750

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Context Info
Confidence 0.08
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.34
Pain Relevance 0.86

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

peptidase activity (Naalad2) cellular_component (Naalad2)
Anatomy Link Frequency
brain 2
Naalad2 (Mus musculus)
Pain Link Frequency Relevance Heat
analgesia 17 98.48 Very High Very High Very High
Eae 14 97.40 Very High Very High Very High
antagonist 6 94.64 High High
Glutamate receptor 5 92.56 High High
intrathecal 2 84.24 Quite High
Central nervous system 4 83.44 Quite High
Neurotransmitter 1 78.92 Quite High
Intracerebroventricular 15 75.00 Quite High
Analgesic 12 75.00 Quite High
Pain 19 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Inflammatory Pain 12 97.40 Very High Very High Very High
Pain 19 5.00 Very Low Very Low Very Low
INFLAMMATION 5 5.00 Very Low Very Low Very Low
Neuropathic Pain 4 5.00 Very Low Very Low Very Low
Nociception 3 5.00 Very Low Very Low Very Low
Bone Cancer 2 5.00 Very Low Very Low Very Low
Urological Neuroanatomy 2 5.00 Very Low Very Low Very Low
Overdose 1 5.00 Very Low Very Low Very Low
Toxicity 1 5.00 Very Low Very Low Very Low
Akathisia 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This research aimed at determining if analgesia obtained following systemic administration of NAAG peptidase inhibitors is due to NAAG activation of group II mGluRs in brain circuits that mediate perception of inflammatory pain.
Positive_regulation (due) of Positive_regulation (activation) of NAAG in brain associated with eae and analgesia
1) Confidence 0.08 Published 2008 Journal Mol Pain Section Abstract Doc Link PMC2517065 Disease Relevance 0.34 Pain Relevance 0.86

General Comments

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