INT243678

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Context Info
Confidence 0.03
First Reported 2008
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 2.84
Pain Relevance 0.83

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (GSR) mitochondrion (GSR) small molecule metabolic process (GSR)
oxidoreductase activity (GSR) transferase activity, transferring glycosyl groups (Ugcg) Golgi apparatus (Ugcg)
GSR (Homo sapiens)
Ugcg (Rattus norvegicus)
Pain Link Frequency Relevance Heat
cytokine 24 99.72 Very High Very High Very High
Inflammatory mediators 1 98.88 Very High Very High Very High
Inflammation 17 96.24 Very High Very High Very High
corticosteroid 2 90.64 High High
cINOD 1 78.24 Quite High
psoriasis 1 71.36 Quite High
Multiple sclerosis 11 70.88 Quite High
rheumatoid arthritis 5 69.36 Quite High
Arthritis 4 59.48 Quite High
Inflammatory response 1 37.16 Quite Low
Disease Link Frequency Relevance Heat
INFLAMMATION 18 98.48 Very High Very High Very High
Adhesions 1 96.64 Very High Very High Very High
Stroke 27 94.20 High High
Stress 51 90.00 High High
Death 2 89.28 High High
Injury 1 88.52 High High
Graft Vs Host Disease 1 84.28 Quite High
Autoimmune Disease 13 82.92 Quite High
Shock 1 81.68 Quite High
Apoptosis 1 71.60 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The mechanism of action of GCs is through its binding with the type I mineralocorticoid receptor (MR) and the type II GR [45].
GR Binding (binding) of GCs
1) Confidence 0.03 Published 2010 Journal Exp Transl Stroke Med Section Body Doc Link PMC2954925 Disease Relevance 1.16 Pain Relevance 0.16
GCs exert most, if not all, of their effects through binding to the glucocorticoid receptor (GR), a ligand-activated transcription factor [12,13].
GR Binding (binding) of GCs
2) Confidence 0.00 Published 2008 Journal BMC Immunol Section Body Doc Link PMC2525626 Disease Relevance 1.69 Pain Relevance 0.68

General Comments

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