From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.05
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 0.37
Pain Relevance 1.16

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Itpr1) nuclear envelope (Itpr1) endoplasmic reticulum (Itpr1)
protein complex (Itpr1) transmembrane transport (Itpr1) cytoplasm (Itpr1)
Anatomy Link Frequency
neurons 1
reticulum 1
Itpr1 (Mus musculus)
Pain Link Frequency Relevance Heat
qutenza 74 99.72 Very High Very High Very High
bradykinin 6 97.12 Very High Very High Very High
antagonist 31 96.56 Very High Very High Very High
Deafferentation 1 80.72 Quite High
cOX1 2 77.24 Quite High
Glutamate 44 67.20 Quite High
calcitonin gene related peptide 14 67.04 Quite High
Pain 30 62.00 Quite High
long-term potentiation 2 61.60 Quite High
Inflammation 19 45.68 Quite Low
Disease Link Frequency Relevance Heat
Targeted Disruption 12 99.96 Very High Very High Very High
Pain 38 62.00 Quite High
Nociception 16 60.68 Quite High
Hemorrhage 2 48.60 Quite Low
INFLAMMATION 21 45.68 Quite Low
Colitis 2 35.28 Quite Low
Pancreatitis 2 31.76 Quite Low
Clostridium Difficile Infection 1 30.16 Quite Low
Increased Venous Pressure Under Development 6 23.36 Low Low
Ganglion Cysts 2 16.88 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Interestingly, capsaicin-induced acid secretion was blocked by capsazepine, L-NAME, indomethacin, in IP receptor knock-out mice and ablation of TRPV1 containing neurons but not by the BK 2 receptor antagonist or the EP1 receptor antagonist [6].
Localization (secretion) of IP receptor in neurons associated with targeted disruption, qutenza, antagonist and bradykinin
1) Confidence 0.05 Published 2008 Journal Current Neuropharmacology Section Body Doc Link PMC2647151 Disease Relevance 0.37 Pain Relevance 1.09
In addition, caffeine also acts on the other primary endoplasmic reticulum calcium release channel, the inositol triphosphate receptor (IP3R), over the same concentration range.
Localization (release) of inositol triphosphate receptor in reticulum
2) Confidence 0.04 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2525814 Disease Relevance 0 Pain Relevance 0.06

General Comments

This test has worked.

Personal tools