INT244158

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Context Info
Confidence 0.56
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0
Pain Relevance 0.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (NOD1) signal transduction (NOD1) plasma membrane (NOD1)
intracellular (NOD1) cytoplasm (NOD1)
Anatomy Link Frequency
T cells 2
NOD1 (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 28 57.44 Quite High
Inflammation 16 5.00 Very Low Very Low Very Low
agonist 6 5.00 Very Low Very Low Very Low
Inflammatory response 6 5.00 Very Low Very Low Very Low
Inflammatory mediators 2 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low
antagonist 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 16 41.48 Quite Low
INFLAMMATION 24 5.00 Very Low Very Low Very Low
Infection 10 5.00 Very Low Very Low Very Low
Injury 6 5.00 Very Low Very Low Very Low
Hyperplasia 2 5.00 Very Low Very Low Very Low
Uveitis 2 5.00 Very Low Very Low Very Low
Toxicity 2 5.00 Very Low Very Low Very Low
Death 2 5.00 Very Low Very Low Very Low
Wrinkles 2 5.00 Very Low Very Low Very Low
Viral Infection 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Effect of UV-B irradiation on the expression of Nod subtypes in HCE-T cells
Regulation (Effect) of Gene_expression (expression) of Nod in T cells
1) Confidence 0.56 Published 2008 Journal Molecular Vision Section Body Doc Link PMC2526096 Disease Relevance 0 Pain Relevance 0
We also wanted to know how the expression of the Nod subfamily, the other sensor subfamily of the NLR family, would be influenced by the UV-B radiation at early (6 h) and late (24 h) time points.
Regulation (influenced) of Gene_expression (expression) of Nod
2) Confidence 0.49 Published 2008 Journal Molecular Vision Section Body Doc Link PMC2526096 Disease Relevance 0 Pain Relevance 0.03

General Comments

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