INT245556

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Context Info
Confidence 0.56
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.76
Pain Relevance 0.29

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Ret) nucleus (Runx1) DNA binding (Runx1)
kinase activity (Ret) transcription factor binding (Runx1)
Anatomy Link Frequency
neurons 1
Runx1 (Mus musculus)
Ret (Mus musculus)
Pain Link Frequency Relevance Heat
dorsal root ganglion 55 99.84 Very High Very High Very High
Dorsal horn 1 24.56 Low Low
Spinal cord 10 19.56 Low Low
Central nervous system 2 5.00 Very Low Very Low Very Low
Peripheral nervous system 2 5.00 Very Low Very Low Very Low
Neuropeptide 2 5.00 Very Low Very Low Very Low
trigeminal ganglion 1 5.00 Very Low Very Low Very Low
Root ganglion neuron 1 5.00 Very Low Very Low Very Low
nociceptor 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 58 99.84 Very High Very High Very High
Targeted Disruption 17 98.02 Very High Very High Very High
Nociception 12 35.96 Quite Low
Neurodegenerative Disease 4 5.00 Very Low Very Low Very Low
Repression 3 5.00 Very Low Very Low Very Low
Cancer 3 5.00 Very Low Very Low Very Low
Death 2 5.00 Very Low Very Low Very Low
Leukemia 2 5.00 Very Low Very Low Very Low
Sprains And Strains 2 5.00 Very Low Very Low Very Low
Hematological Disease 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In Ret conditional knockout DRG, Runx1 expression is normal but a part of Runx1 target genes are affected, suggesting the GFR/Ret dependent transcriptional regulation by Runx1 in DRG neurons [42].
Runx1 Regulation (regulation) of Ret in neurons associated with targeted disruption and dorsal root ganglion
1) Confidence 0.56 Published 2008 Journal Neural Develop Section Body Doc Link PMC2531103 Disease Relevance 0.76 Pain Relevance 0.29

General Comments

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