INT245585

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Context Info
Confidence 0.31
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.19
Pain Relevance 0.10

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

phosphatase activity (Ptpn4) cytoskeletal protein binding (Ptpn4) plasma membrane (Ptpn4)
cytoskeleton (Ptpn4) cytoplasm (Ptpn4)
Anatomy Link Frequency
motor nerve 1
Ptpn4 (Mus musculus)
Pain Link Frequency Relevance Heat
Sciatic nerve 1 99.56 Very High Very High Very High
nMDA receptor 1 96.56 Very High Very High Very High
sodium channel 2 20.32 Low Low
Hippocampus 12 5.00 Very Low Very Low Very Low
Thalamus 8 5.00 Very Low Very Low Very Low
cerebral cortex 5 5.00 Very Low Very Low Very Low
Eae 4 5.00 Very Low Very Low Very Low
Pyramidal cell 3 5.00 Very Low Very Low Very Low
Spinal cord 2 5.00 Very Low Very Low Very Low
midbrain 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cognitive Disorder 10 81.60 Quite High
Apoptosis 1 59.12 Quite High
Targeted Disruption 44 50.00 Quite Low
Anxiety Disorder 8 5.00 Very Low Very Low Very Low
Overdose 2 5.00 Very Low Very Low Very Low
Body Weight 2 5.00 Very Low Very Low Very Low
Nociception 2 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Disease 1 5.00 Very Low Very Low Very Low
Urological Neuroanatomy 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
PTPBL-KO (non receptor like PTP-NRPTP) mice display impaired motor nerve repair in a model of sciatic nerve crush lesion [8] and PTPMEG (NRPTP) interacts with key intracellular players leading to the stimulation of the channel activity of NMDA receptors [9].
PTPMEG Binding (interacts) of in motor nerve associated with nmda receptor and sciatic nerve
1) Confidence 0.31 Published 2008 Journal Behav Brain Funct Section Body Doc Link PMC2531118 Disease Relevance 0.19 Pain Relevance 0.10

General Comments

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