INT246029

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Context Info
Confidence 0.21
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 2.83
Pain Relevance 0.35

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (IL23R)
IL23R (Homo sapiens)
Pain Link Frequency Relevance Heat
Crohn's disease 37 99.72 Very High Very High Very High
Bile 2 73.76 Quite High
Inflammation 13 72.64 Quite High
Inflammatory mediators 1 69.92 Quite High
cytokine 1 5.00 Very Low Very Low Very Low
psoriasis 1 5.00 Very Low Very Low Very Low
spinal inflammation 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 57 99.72 Very High Very High Very High
Genetic Predisposition To Disease 3 99.60 Very High Very High Very High
Inflammatory Bowel Disease 21 96.84 Very High Very High Very High
Biliary Liver Cirrhosis 15 96.28 Very High Very High Very High
Primary Sclerosing Cholangitis 21 95.84 Very High Very High Very High
Disease Susceptibility 3 94.32 High High
Autoimmune Disease 2 91.00 High High
Liver Disease 2 84.92 Quite High
INFLAMMATION 11 72.64 Quite High
Mucositis 1 63.12 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
It has been reported that the genetic predisposition to CD is caused by alterations in several genes, including NOD2/CARD15, OCTN1, OCTN2, DLG, ATG16L1 and IL23R [11-14].
Regulation (alterations) of IL23R associated with crohn's disease and genetic predisposition to disease
1) Confidence 0.21 Published 2008 Journal BMC Med Genet Section Body Doc Link PMC2535589 Disease Relevance 2.83 Pain Relevance 0.35

General Comments

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