INT246795

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Context Info
Confidence 0.05
First Reported 2008
Last Reported 2008
Negated 0
Speculated 1
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (Ogg1) cell differentiation (Ros1) mitochondrion (Ogg1)
lyase activity (Ogg1) cytoplasm (Ogg1) cell proliferation (Ros1)
Ros1 (Rattus norvegicus)
Ogg1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
agonist 2 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Diabetes Mellitus 78 50.00 Quite Low
Stress 14 5.00 Very Low Very Low Very Low
Hyperglycemia 12 5.00 Very Low Very Low Very Low
Injury 8 5.00 Very Low Very Low Very Low
Apoptosis 4 5.00 Very Low Very Low Very Low
Body Weight 4 5.00 Very Low Very Low Very Low
Renal Disease 4 5.00 Very Low Very Low Very Low
Targeted Disruption 2 5.00 Very Low Very Low Very Low
Death 2 5.00 Very Low Very Low Very Low
Adhesions 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We next investigated a potential role for ROS in Akt and tuberin phosphorylation and downregulation of OGG1.
ROS Spec (investigated) Positive_regulation (role) of Phosphorylation (phosphorylation) of OGG1
1) Confidence 0.05 Published 2008 Journal Diabetes Section Body Doc Link PMC2551671 Disease Relevance 0 Pain Relevance 0
These data indicate that ROS are critical for high glucoseĀ–induced Akt and tuberin phosphorylation and downregulation of OGG1.
ROS Positive_regulation (critical) of Phosphorylation (phosphorylation) of OGG1
2) Confidence 0.04 Published 2008 Journal Diabetes Section Body Doc Link PMC2551671 Disease Relevance 0 Pain Relevance 0

General Comments

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