INT246803

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Context Info
Confidence 0.14
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.85
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Akt1, Tsc2) cytosol (Akt1, Tsc2) signal transduction (Akt1, Tsc2)
nucleus (Akt1, Tsc2) nucleoplasm (Akt1) transport (Akt1)
Anatomy Link Frequency
kidney cortex 1
Akt1 (Rattus norvegicus)
Tsc2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
agonist 2 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Diabetes Mellitus 78 99.82 Very High Very High Very High
Injury 8 96.72 Very High Very High Very High
Apoptosis 4 91.96 High High
Stress 14 64.64 Quite High
Death 2 28.16 Quite Low
Hyperglycemia 12 5.00 Very Low Very Low Very Low
Body Weight 4 5.00 Very Low Very Low Very Low
Renal Disease 4 5.00 Very Low Very Low Very Low
Targeted Disruption 2 5.00 Very Low Very Low Very Low
Adhesions 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We also found that high glucoseĀ–induced tuberin phosphorylation by Akt is associated with biological consequences, namely a decrease in OGG1 expression and increased 8-oxodG levels.
Akt Binding (associated) of tuberin
1) Confidence 0.14 Published 2008 Journal Diabetes Section Body Doc Link PMC2551671 Disease Relevance 0.19 Pain Relevance 0
We also demonstrate that diabetes-induced Akt and tuberin phosphorylation are associated with a decrease in OGG1 expression and accumulation of 8-oxodG in kidney cortex of diabetic rats.
Akt Binding (associated) of tuberin in kidney cortex associated with diabetes mellitus
2) Confidence 0.14 Published 2008 Journal Diabetes Section Body Doc Link PMC2551671 Disease Relevance 0.66 Pain Relevance 0

General Comments

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