INT247031

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Context Info
Confidence 0.00
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Npr1, Prkaca) nucleoplasm (Prkaca) mitochondrion (Prkaca)
Golgi apparatus (Prkaca) intracellular (Npr1) protein complex (Prkaca)
Npr1 (Rattus norvegicus)
Prkaca (Rattus norvegicus)
Pain Link Frequency Relevance Heat
agonist 2 5.00 Very Low Very Low Very Low
fibrosis 2 5.00 Very Low Very Low Very Low
Piles 1 5.00 Very Low Very Low Very Low
headache 1 5.00 Very Low Very Low Very Low
cva 1 5.00 Very Low Very Low Very Low
5HT 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Constipation 39 50.00 Quite Low
Body Weight 4 5.00 Very Low Very Low Very Low
Diarrhoea 3 5.00 Very Low Very Low Very Low
Spasticity 2 5.00 Very Low Very Low Very Low
Cystic Fibrosis 2 5.00 Very Low Very Low Very Low
Cardiovascular Disease 1 5.00 Very Low Very Low Very Low
Anorectal Disorders 1 5.00 Very Low Very Low Very Low
Cv General 3 Under Development 1 5.00 Very Low Very Low Very Low
Vomiting 1 5.00 Very Low Very Low Very Low
Enteropathy 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
It is therefore that the ISC response to NAR was most likely mediated by intracellular cAMP production generated by adenylate cyclase, which in turn activates PKA and subsequently the apical cAMP-dependent chloride channel that was probably CFTR.
cyclase Positive_regulation (activates) of PKA
1) Confidence 0.00 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2553183 Disease Relevance 0 Pain Relevance 0

General Comments

This test has worked.

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