INT247971

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Context Info
Confidence 0.13
First Reported 2008
Last Reported 2010
Negated 1
Speculated 1
Reported most in Body
Documents 4
Total Number 7
Disease Relevance 6.41
Pain Relevance 1.50

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
anterior 1
external 1
body 1
MAFD1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Pain 161 98.98 Very High Very High Very High
Neuropathic pain 104 92.72 High High
Central nervous system 15 92.12 High High
primary somatosensory cortex 58 78.24 Quite High
insula 54 76.80 Quite High
addiction 20 46.32 Quite Low
Somatosensory cortex 36 39.76 Quite Low
Anterior cingulate cortex 2 37.00 Quite Low
fifth nerve 26 20.12 Low Low
Lasting pain 50 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Mental Disorders 345 100.00 Very High Very High Very High
Munchausen Syndrome 35 100.00 Very High Very High Very High
Syndrome 63 99.58 Very High Very High Very High
Pain 199 98.98 Very High Very High Very High
Disease 20 98.28 Very High Very High Very High
Scoliosis 314 97.66 Very High Very High Very High
Congenital Anomalies 14 97.48 Very High Very High Very High
Somatoform Disorder 6 93.72 High High
Neuropathic Pain 140 92.72 High High
Unconsciousness 2 66.64 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
As summarized in Table 2, we analyzed the correlation of the duration of the disorder with: 1) the absolute values of positive (activation) and negative (deactivation) BOLD signals (Z-values) during allodynic stimulation under the cluster areas that showed cortical thickness changes (TNP vs.
Spec (analyzed) Gene_expression (correlation) of duration of the disorder associated with neuropathic pain
1) Confidence 0.13 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2561059 Disease Relevance 0.71 Pain Relevance 0.61
In the anterior insular cortex contralateral to the pain there was a positive correlation between cortical thickness and the duration of the disorder (p<0.04), which does not corroborate with the general results since this ROI showed thinning when compared to healthy controls.


Gene_expression (thickness) of duration of the disorder in anterior associated with pain
2) Confidence 0.13 Published 2008 Journal PLoS ONE Section Body Doc Link PMC2561059 Disease Relevance 0.62 Pain Relevance 0.80
Free will and mental disorder: Exploring the relationship

A link between mental disorder and freedom is clearly present in the introduction of the fourth edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-IV).

Gene_expression (present) of mental disorder associated with mental disorders
3) Confidence 0.06 Published 2010 Journal Theor Med Bioeth Section Title Doc Link PMC2975916 Disease Relevance 0.88 Pain Relevance 0
I examine two domains in which a connection between mental disorder and free will is present: the philosophy of free will and forensic psychiatry.
Gene_expression (connection) of mental disorder associated with mental disorders
4) Confidence 0.05 Published 2010 Journal Theor Med Bioeth Section Abstract Doc Link PMC2975916 Disease Relevance 0.89 Pain Relevance 0
Free will and mental disorder: Exploring the relationship

A link between mental disorder and freedom is clearly present in the introduction of the fourth edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-IV).

Gene_expression (present) of mental disorder associated with mental disorders
5) Confidence 0.05 Published 2010 Journal Theor Med Bioeth Section Title Doc Link PMC2975916 Disease Relevance 0.88 Pain Relevance 0
In psychiatric classification Munchausen's syndrome is a severe form of factitious disorder, where symptoms (physical or psychiatric) are produced deliberately without evidence of external incentives [19].
Neg (without) Gene_expression (produced) of factitious disorder in external associated with syndrome and munchausen syndrome
6) Confidence 0.03 Published 2008 Journal Cases J Section Body Doc Link PMC2572607 Disease Relevance 1.50 Pain Relevance 0
Neurological abnormalities with AIS have been explained by four fairly comprehensive concepts for pathogenesis:

(1) visuo-spatial perceptual impairment producing a motor control problem [104]; (2) body-spatial orientation concept [69]; (3) neurodevelopmental concept [105,106]; and (4) sensory integration disorder [102].

Gene_expression (producing) of sensory integration disorder in body associated with scoliosis and congenital anomalies
7) Confidence 0.01 Published 2009 Journal Scoliosis Section Body Doc Link PMC2781798 Disease Relevance 0.94 Pain Relevance 0.09

General Comments

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