INT249198

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Context Info
Confidence 0.16
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.16
Pain Relevance 0.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

lyase activity (CA2) response to stress (CA2) cytoplasm (CA2)
cytosol (CA2) signal transduction (CD38) extracellular space (CA2)
CD38 (Homo sapiens)
CA2 (Homo sapiens)
Pain Link Frequency Relevance Heat
antagonist 4 94.04 High High
Inflammation 16 5.00 Very Low Very Low Very Low
agonist 8 5.00 Very Low Very Low Very Low
addiction 6 5.00 Very Low Very Low Very Low
TRP channel 4 5.00 Very Low Very Low Very Low
Inflammatory response 4 5.00 Very Low Very Low Very Low
cytokine 2 5.00 Very Low Very Low Very Low
cINOD 2 5.00 Very Low Very Low Very Low
fibrosis 2 5.00 Very Low Very Low Very Low
Inflammatory mediators 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 6 95.52 Very High Very High Very High
INFLAMMATION 22 5.00 Very Low Very Low Very Low
Adhesions 10 5.00 Very Low Very Low Very Low
Disease 10 5.00 Very Low Very Low Very Low
Pulmonary Disease 4 5.00 Very Low Very Low Very Low
Neutrophil Disorders 4 5.00 Very Low Very Low Very Low
Stress 4 5.00 Very Low Very Low Very Low
Respiratory Syncytial Virus 2 5.00 Very Low Very Low Very Low
Toxicity 2 5.00 Very Low Very Low Very Low
Prostate Cancer 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Based on these observations and additional experiments, Partida-Sanchez et al. [81] postulated that cADPr directly activates Ca2+ influx by mobilizing the intracellular Ca2+ store through the ryanodine receptor.
cADPr Positive_regulation (activates) of Ca2
1) Confidence 0.16 Published 2008 Journal J Leukoc Biol Section Body Doc Link PMC2567897 Disease Relevance 0.10 Pain Relevance 0.05
There is no consensus about mechanisms by which cADPr induces Ca2+ influx, but the possibility that cADPr is first hydrolyzed to ADPr [86] and then gates channels has recently attracted attention.
cADPr Positive_regulation (induces) of Ca2
2) Confidence 0.14 Published 2008 Journal J Leukoc Biol Section Body Doc Link PMC2567897 Disease Relevance 0.07 Pain Relevance 0.03

General Comments

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