INT249303

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Context Info
Confidence 0.41
First Reported 2008
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 7
Total Number 7
Disease Relevance 1.12
Pain Relevance 0.93

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Preb) vesicle-mediated transport (Preb) endoplasmic reticulum (Preb)
nucleus (Preb) DNA binding (Preb)
Anatomy Link Frequency
neurons 2
Preb (Mus musculus)
Pain Link Frequency Relevance Heat
depression 103 98.96 Very High Very High Very High
medulla 92 93.68 High High
gABA 37 87.16 High High
adenocard 66 86.00 High High
agonist 30 84.32 Quite High
Neuronal excitability 30 82.16 Quite High
antagonist 49 74.64 Quite High
Spinal cord 37 37.44 Quite Low
Raphe 6 33.12 Quite Low
imagery 20 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Depression 109 98.96 Very High Very High Very High
Death 1 89.96 High High
Hypoxia 30 73.36 Quite High
Convulsion 36 70.72 Quite High
Targeted Disruption 6 57.28 Quite High
Rheumatoid Arthritis 18 5.00 Very Low Very Low Very Low
Stress 12 5.00 Very Low Very Low Very Low
Wrinkles 1 5.00 Very Low Very Low Very Low
Nociception 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
While glycinergic neurons seem to be located in the area of the preBötC [40], their role in inspiratory network function in the isolated preBötC is not clear.
Regulation (role) of preB in neurons
1) Confidence 0.41 Published 2009 Journal Pflugers Arch Section Body Doc Link PMC2691554 Disease Relevance 0.24 Pain Relevance 0.09
Effects of A1R agonism/antagonism on preBötC population activity in slice preparations
Regulation (Effects) of preB
2) Confidence 0.39 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2567986 Disease Relevance 0.17 Pain Relevance 0.25
Effects of A1R agonism/antagonism on preBötC population activity in island preparations
Regulation (Effects) of preB
3) Confidence 0.39 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2567986 Disease Relevance 0.21 Pain Relevance 0
Modulation of preBötC rhythmogenesis represents a central focus of research into this region's function.
Regulation (Modulation) of preB
4) Confidence 0.23 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2567986 Disease Relevance 0.08 Pain Relevance 0.19
Modulation of at least some of these inputs, such as the contralateral preBötC, can affect the pattern of preBötC output [10,34].
Regulation (Modulation) of preB
5) Confidence 0.23 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2567986 Disease Relevance 0.16 Pain Relevance 0.08
Accordingly, modulation of preBötC rhythmogenesis likely involves regulation of multiple aspects of network function, including the modulation of membrane properties and of synaptic interactions [6,11-13].
Regulation (modulation) of preB
6) Confidence 0.23 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2567986 Disease Relevance 0.21 Pain Relevance 0.32
These findings, are consistent with the notion that the modulatory effects of A1R activation, although likely to integrate with concurrent synaptic inputs, are greater in their overall effect on preBötC activity than are A1R-induced changes in the net synaptic input to preBötC neurons from sources originating outside the preBötC.
Regulation (effect) of preB in neurons
7) Confidence 0.23 Published 2008 Journal BMC Neurosci Section Body Doc Link PMC2567986 Disease Relevance 0.06 Pain Relevance 0

General Comments

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