INT249507

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.38
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.58
Pain Relevance 0.55

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

endosome (Ephb1) signal transduction (Ephb1) plasma membrane (Ephb1)
cytoplasm (Ephb1)
Anatomy Link Frequency
synapses 2
Ephb1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Neuropathic pain 9 99.84 Very High Very High Very High
Hyperalgesia 11 98.08 Very High Very High Very High
Spinal cord 30 92.36 High High
nMDA receptor 28 75.24 Quite High
Root ganglion neuron 3 74.48 Quite High
Dorsal horn 11 66.32 Quite High
allodynia 7 56.24 Quite High
Inflammation 9 50.00 Quite Low
Eae 10 44.24 Quite Low
dorsal rhizotomy 1 39.52 Quite Low
Disease Link Frequency Relevance Heat
Neuropathic Pain 18 99.84 Very High Very High Very High
Nervous System Injury 3 99.84 Very High Very High Very High
Hyperalgesia 25 98.08 Very High Very High Very High
Targeted Disruption 2 82.96 Quite High
Ganglion Cysts 5 74.08 Quite High
INFLAMMATION 9 50.00 Quite Low
Injury 5 42.56 Quite Low
Nociception 14 5.00 Very Low Very Low Very Low
Pain 13 5.00 Very Low Very Low Very Low
Inflammatory Pain 7 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
A role for EphB1-ephrinB2 interactions in neuropathic pain has recently been identified, providing further support to the hypothesis that this receptor is the main contributor to synaptic plasticity of excitatory synapses in the spinal cord (Kobayashi et al., 2007; Song et al., 2008a,b).
Regulation (role) of EphB1-ephrinB2 Binding (interactions) of in synapses associated with neuropathic pain and spinal cord
1) Confidence 0.38 Published 2008 Journal Neuroscience Section Body Doc Link PMC2568875 Disease Relevance 0.58 Pain Relevance 0.55

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox