INT250043

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Context Info
Confidence 0.29
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.56
Pain Relevance 0.40

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Gpr143) Golgi apparatus (Gpr143) plasma membrane (Gpr143)
lysosome (Gpr143) signal transducer activity (Gpr143)
Gpr143 (Mus musculus)
Pain Link Frequency Relevance Heat
Anterior cingulate cortex 64 92.04 High High
Pyramidal cell 12 87.68 High High
nMDA receptor antagonist 2 84.16 Quite High
nMDA receptor 5 78.24 Quite High
Lasting pain 24 75.68 Quite High
addiction 3 64.16 Quite High
Glutamate 31 50.00 Quite Low
IPN 11 50.00 Quite Low
antagonist 1 47.08 Quite Low
Neuronal excitability 3 9.28 Low Low
Disease Link Frequency Relevance Heat
Targeted Disruption 31 96.76 Very High Very High Very High
Pain 34 75.68 Quite High
Inflammatory Pain 11 50.00 Quite Low
INFLAMMATION 8 5.00 Very Low Very Low Very Low
Anxiety Disorder 2 5.00 Very Low Very Low Very Low
Nociception 2 5.00 Very Low Very Low Very Low
Depression 1 5.00 Very Low Very Low Very Low
Drug Dependence 1 5.00 Very Low Very Low Very Low
Disseminated Intravascular Coagulation 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
It is well known that Ap oa1 is selectively coupled to Gs protein [38] and activation of heterologous expression of Ap oa1 in HEK293 cells selectively stimulated cAMP synthesis after octopamine application [32].
Positive_regulation (activation) of Gene_expression (expression) of oa1
1) Confidence 0.29 Published 2008 Journal Mol Pain Section Body Doc Link PMC2570662 Disease Relevance 0.56 Pain Relevance 0.40

General Comments

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