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Context Info
Confidence 0.04
First Reported 2008
Last Reported 2008
Negated 1
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.52
Pain Relevance 0.59

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Gpr143) Golgi apparatus (Gpr143) nucleus (Spdya)
plasma membrane (Gpr143) cell cycle (Spdya) lysosome (Gpr143)
Gpr143 (Mus musculus)
Spdya (Mus musculus)
Pain Link Frequency Relevance Heat
Anterior cingulate cortex 64 88.36 High High
Lasting pain 24 85.84 High High
spinal dorsal horn 1 85.20 High High
Neuronal excitability 3 80.56 Quite High
Glutamate 31 71.96 Quite High
IPN 11 50.00 Quite Low
Acute pain 3 43.52 Quite Low
tail-flick 5 34.48 Quite Low
nMDA receptor 5 27.44 Quite Low
Inflammatory response 1 23.36 Low Low
Disease Link Frequency Relevance Heat
Targeted Disruption 31 99.66 Very High Very High Very High
Pain 34 85.84 High High
Inflammatory Pain 11 50.00 Quite Low
Nociception 2 29.76 Quite Low
INFLAMMATION 8 22.96 Low Low
Anxiety Disorder 2 5.00 Very Low Very Low Very Low
Depression 1 5.00 Very Low Very Low Very Low
Drug Dependence 1 5.00 Very Low Very Low Very Low
Disseminated Intravascular Coagulation 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Another advantage for using the transgenic mice is that Ap oa1 receptor is Aplysia Gs protein-coupled receptor and is thus not activated endogenously.
Neg (not) Positive_regulation (activated) of oa1 Binding (receptor) of Gs protein associated with targeted disruption
1) Confidence 0.04 Published 2008 Journal Mol Pain Section Body Doc Link PMC2570662 Disease Relevance 0.52 Pain Relevance 0.59

General Comments

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