INT250076

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Context Info
Confidence 0.10
First Reported 2008
Last Reported 2008
Negated 0
Speculated 1
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.06
Pain Relevance 0.35

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Grin2a) plasma membrane (Grin2a) locomotion (Grin2a)
Anatomy Link Frequency
spike 2
Grin2a (Rattus norvegicus)
Ltp (Mus musculus)
Pain Link Frequency Relevance Heat
long-term potentiation 74 100.00 Very High Very High Very High
Action potential 3 98.24 Very High Very High Very High
Pyramidal cell 3 83.44 Quite High
Hippocampus 10 81.28 Quite High
antagonist 17 57.68 Quite High
nMDA receptor 4 50.00 Quite Low
amygdala 8 5.00 Very Low Very Low Very Low
imagery 6 5.00 Very Low Very Low Very Low
isoflurane 3 5.00 Very Low Very Low Very Low
Anterior cingulate cortex 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Anxiety Disorder 47 55.84 Quite High
Shock 1 23.24 Low Low
Pain 1 5.00 Very Low Very Low Very Low
Targeted Disruption 1 5.00 Very Low Very Low Very Low
Anaerobic Bacterial Infections 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
To test whether the involvement of NR2B-NMDARs is dependent on specific LTP induction paradigm, we first examined the role of NR2B- and NR2A-NMDARs in LTP induced by spike-timing protocol (also named EPSPs-APs protocol) (Figure 1A).
NR2A-NMDARs Spec (examined) Regulation (role) of Positive_regulation (induced) of LTP in spike associated with action potential and long-term potentiation
1) Confidence 0.10 Published 2008 Journal Mol Brain Section Body Doc Link PMC2570668 Disease Relevance 0.06 Pain Relevance 0.35

General Comments

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