INT250461

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.01
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.26

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Car2) extracellular space (Car2) lyase activity (Car2)
cytoplasm (Car2)
Anatomy Link Frequency
reticulum 1
Car2 (Mus musculus)
Ba2 (Mus musculus)
Pain Link Frequency Relevance Heat
nMDA receptor 66 95.52 Very High Very High Very High
agonist 4 76.28 Quite High
substance P 11 50.00 Quite Low
addiction 1 39.72 Quite Low
Dorsal horn neuron 12 34.32 Quite Low
antagonist 14 21.76 Low Low
Spinal cord 12 5.00 Very Low Very Low Very Low
Dorsal horn 10 5.00 Very Low Very Low Very Low
allodynia 8 5.00 Very Low Very Low Very Low
Hyperalgesia 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Pain 8 5.00 Very Low Very Low Very Low
Neuropathic Pain 8 5.00 Very Low Very Low Very Low
Hyperalgesia 5 5.00 Very Low Very Low Very Low
Hypersensitivity 4 5.00 Very Low Very Low Very Low
Targeted Disruption 3 5.00 Very Low Very Low Very Low
Injury 2 5.00 Very Low Very Low Very Low
Nociception 1 5.00 Very Low Very Low Very Low
Disseminated Intravascular Coagulation 1 5.00 Very Low Very Low Very Low
Drug Induced Neurotoxicity 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Although most of the extracellular Ca2+ was replaced with Ba2+ in these experiments, it was still possible that the remaining bath Ca2+ or Ca2+ released from endoplasmic reticulum (ER) was sufficient to trigger activation of other currents, altering g(-90 mV)/g(MIC) ratio and VMIC values.
Ca2 Binding (replaced) of Ba2 in reticulum
1) Confidence 0.01 Published 2008 Journal Mol Pain Section Body Doc Link PMC2572590 Disease Relevance 0 Pain Relevance 0.26

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox