INT250514

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Context Info
Confidence 0.60
First Reported 2008
Last Reported 2010
Negated 1
Speculated 0
Reported most in Body
Documents 2
Total Number 4
Disease Relevance 0.85
Pain Relevance 0.26

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Pou1f1) DNA binding (Pou1f1) transcription factor binding (Pou1f1)
cytoplasm (Pou1f1)
Anatomy Link Frequency
neurons 1
Pit 1
Pou1f1 (Mus musculus)
Pain Link Frequency Relevance Heat
Somatostatin 17 96.08 Very High Very High Very High
Osteoarthritis 3 81.84 Quite High
Central nervous system 7 14.48 Low Low
cva 6 5.00 Very Low Very Low Very Low
anesthesia 5 5.00 Very Low Very Low Very Low
isoflurane 4 5.00 Very Low Very Low Very Low
Inflammation 2 5.00 Very Low Very Low Very Low
tolerance 2 5.00 Very Low Very Low Very Low
cerebral cortex 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Osteoporosis 15 96.76 Very High Very High Very High
Calcification 18 91.52 High High
Apoptosis 42 88.68 High High
Osteoarthritis 3 81.84 Quite High
Embryonic Lethality 24 74.28 Quite High
Death 20 67.64 Quite High
Anaemia 42 54.80 Quite High
Lifespan 33 50.00 Quite Low
Leukemia 3 29.84 Quite Low
Aging 15 7.36 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Expression and activity of PiT1 in chondrogenic cells was found to be regulated by extracellular Pi concentration [16] and transforming growth factor-?
Regulation (regulated) of Gene_expression (Expression) of PiT1
1) Confidence 0.60 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2818845 Disease Relevance 0.42 Pain Relevance 0.04
Interestingly, our results showed that Na+-Pi uptake in PiT1-null MEFs was unaffected (Fig. 8B), with no change in its kinetic properties (Vmax ?
Neg (unaffected) Regulation (unaffected) of Gene_expression (uptake) of PiT1
2) Confidence 0.44 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2818845 Disease Relevance 0.07 Pain Relevance 0
Staining with the proliferation marker Ki67 was less intense in sections of E12.5 PiT1?
Regulation (intense) of Gene_expression (sections) of PiT1
3) Confidence 0.44 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2818845 Disease Relevance 0.36 Pain Relevance 0
Accordingly, Pit-1 expression, which is controlled by GHRH neurons and steers somatotropic cell differentiation, was half normal in mutant pituitaries (Figure 3E).
Regulation (controlled) of in neurons Gene_expression (expression) of Pit-1 in Pit
4) Confidence 0.07 Published 2008 Journal PLoS Biology Section Body Doc Link PMC2573928 Disease Relevance 0 Pain Relevance 0.21

General Comments

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