INT250771

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Context Info
Confidence 0.43
First Reported 2008
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 2.45
Pain Relevance 0.75

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (Atl1) endoplasmic reticulum (Atl1) GTPase activity (Atl1)
Anatomy Link Frequency
epithelial cells 1
kidney 1
Atl1 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 57 95.00 High High
fibrosis 164 88.60 High High
chemokine 22 71.12 Quite High
Inflammatory response 4 52.48 Quite High
Inflammatory mediators 2 40.84 Quite Low
cytokine 18 37.52 Quite Low
antagonist 1 31.44 Quite Low
ketamine 5 5.00 Very Low Very Low Very Low
anesthesia 3 5.00 Very Low Very Low Very Low
addiction 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Fibrosis 102 100.00 Very High Very High Very High
INFLAMMATION 59 95.00 High High
Lung Injury 36 92.72 High High
Adult Respiratory Distress Syndrome 68 88.88 High High
Pulmonary Fibrosis 88 88.60 High High
Renal Disease 10 88.16 High High
Chronic Renal Failure 6 85.60 High High
Cicatrix 7 74.68 Quite High
Ureteral Obstruction 45 73.84 Quite High
Internal Fibrosis 9 70.36 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The expression of S100A4/FSP1 indicates the presence of an ongoing angiogenetic program determining the mesenchymal phenotype [16].
Gene_expression (expression) of FSP1
1) Confidence 0.43 Published 2008 Journal Crit Care Section Body Doc Link PMC2575592 Disease Relevance 0.50 Pain Relevance 0.18
The expression of S100A4/FSP1 in the epithelium indicates the presence of transition from epithelial cells to fibroblasts.
Gene_expression (expression) of FSP1 in epithelial cells
2) Confidence 0.43 Published 2008 Journal Crit Care Section Body Doc Link PMC2575592 Disease Relevance 1.00 Pain Relevance 0.34
It is important to highlight, however, that late treatment with Hemin was able to reduce the expression and production of pro-fibrotic molecules, as well as type I collagen and FSP-1 deposition on the injured kidney.
Gene_expression (expression) of FSP-1 in kidney associated with fibrosis
3) Confidence 0.01 Published 2010 Journal PLoS ONE Section Body Doc Link PMC3001459 Disease Relevance 0.95 Pain Relevance 0.23

General Comments

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