INT251159

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.16
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.87

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Clu, Trpv1) mitochondrion (Clu) endoplasmic reticulum (Clu)
lipid metabolic process (Trpv1) response to stress (Clu) cytoplasm (Clu)
Clu (Rattus norvegicus)
Trpv1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
qutenza 44 99.68 Very High Very High Very High
Kinase C 21 68.40 Quite High
dorsal root ganglion 28 49.68 Quite Low
bradykinin 2 42.72 Quite Low
Serotonin 1 39.36 Quite Low
nociceptor 1 34.72 Quite Low
Inflammation 4 32.24 Quite Low
Hyperalgesia 2 31.20 Quite Low
imagery 9 5.00 Very Low Very Low Very Low
TRP channel 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 28 49.68 Quite Low
INFLAMMATION 4 32.24 Quite Low
Hyperalgesia 3 31.20 Quite Low
Nociception 3 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Aids-related Complex 1 5.00 Very Low Very Low Very Low
Rupture 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Our results consistently support the hypothesis that DAG and its analogs activate TRPV1 directly by binding at the capsaicin-binding site.
DAG Binding (binding) of Positive_regulation (activate) of TRPV1 associated with qutenza
1) Confidence 0.16 Published 2008 Journal Mol Pain Section Body Doc Link PMC2576176 Disease Relevance 0 Pain Relevance 0.87

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox