INT251515

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Context Info
Confidence 0.18
First Reported 2007
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 5
Total Number 5
Disease Relevance 0.99
Pain Relevance 1.06

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (REM1) GTPase activity (REM1)
Anatomy Link Frequency
neurons 2
cortex 2
REM1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Hippocampus 252 100.00 Very High Very High Very High
primary somatosensory cortex 200 100.00 Very High Very High Very High
addiction 4 88.16 High High
cerebral cortex 16 84.56 Quite High
halothane 12 5.00 Very Low Very Low Very Low
long-term potentiation 8 5.00 Very Low Very Low Very Low
ketamine 4 5.00 Very Low Very Low Very Low
Nerve growth factor 4 5.00 Very Low Very Low Very Low
anesthesia 4 5.00 Very Low Very Low Very Low
tolerance 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apnoea 34 100.00 Very High Very High Very High
Aids-related Complex 140 98.72 Very High Very High Very High
Sleep Disorders 8 83.76 Quite High
Sleep Apnea Syndromes 2 51.28 Quite High
Cognitive Disorder 4 46.08 Quite Low
Cicatrix 4 5.00 Very Low Very Low Very Low
Disease 4 5.00 Very Low Very Low Very Low
Lifespan 4 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Targeted Disruption 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
For example, the faster overall decay of the REM bout distributions with OSA was attributed to faster time constants (particularly of the fast exponential) as well as an increase of the relative contribution of the fast component (at the expense of the slow component).
Positive_regulation (attributed) of Gene_expression (decay) of REM associated with apnoea
1) Confidence 0.18 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2893208 Disease Relevance 0.39 Pain Relevance 0
Four hours after experience, IEG expression was specifically upregulated during REM sleep in the cortex, but not in the hippocampus.
Positive_regulation (upregulated) of Gene_expression (sleep) of REM in cortex associated with hippocampus
2) Confidence 0.03 Published 2007 Journal Frontiers in Neuroscience Section Abstract Doc Link PMC2577304 Disease Relevance 0.17 Pain Relevance 0.26
Despite the link between cortical IEG upregulation and post-experience REM sleep, analysis of the relation between IEG expression in S1 and time spent in REM sleep within 60?
Positive_regulation (experience) of Gene_expression (sleep) of REM associated with primary somatosensory cortex
3) Confidence 0.03 Published 2007 Journal Frontiers in Neuroscience Section Body Doc Link PMC2577304 Disease Relevance 0.23 Pain Relevance 0.61
Indeed, IEG reinduction during REM sleep may explain the second wave of arc protein expression detected 8–24?
Positive_regulation (during) of Gene_expression (sleep) of REM associated with aids-related complex
4) Confidence 0.03 Published 2007 Journal Frontiers in Neuroscience Section Body Doc Link PMC2577304 Disease Relevance 0.20 Pain Relevance 0
Next, we compared the mean firing rates of activated neurons in S1, HP, and V1 across WK, SWS, and REM sleep occurring between 2?
Positive_regulation (activated) of Gene_expression (neurons) of REM in neurons associated with primary somatosensory cortex and hippocampus
5) Confidence 0.03 Published 2007 Journal Frontiers in Neuroscience Section Body Doc Link PMC2577304 Disease Relevance 0 Pain Relevance 0.18

General Comments

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