INT251623

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Context Info
Confidence 0.02
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.31
Pain Relevance 0.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

endosome (Fig4) lyase activity (Car2) Golgi apparatus (Fig4)
endoplasmic reticulum (Fig4) cytoplasm (Car2) pigmentation (Fig4)
Car2 (Mus musculus)
Fig4 (Mus musculus)
Pain Link Frequency Relevance Heat
antagonist 1 59.44 Quite High
imagery 6 55.32 Quite High
Glutamate 29 54.32 Quite High
Nortriptyline 1 33.92 Quite Low
Desipramine 1 32.72 Quite Low
Serotonin 8 23.04 Low Low
Dopamine 4 21.88 Low Low
nMDA receptor 5 5.00 Very Low Very Low Very Low
Antihistamine 2 5.00 Very Low Very Low Very Low
Potency 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 56 91.44 High High
Toxicity 4 53.52 Quite High
Apoptosis 6 5.00 Very Low Very Low Very Low
Myelodysplastic Syndromes 4 5.00 Very Low Very Low Very Low
Targeted Disruption 2 5.00 Very Low Very Low Very Low
Congenital Anomalies 2 5.00 Very Low Very Low Very Low
Cognitive Disorder 2 5.00 Very Low Very Low Very Low
Neurodegenerative Disease 2 5.00 Very Low Very Low Very Low
Sprains And Strains 1 5.00 Very Low Very Low Very Low
Disorders Of Creatine Metabolism 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Thus, neuroprotective effects of Dimebon observed in our experiments (Table 1, Fig 5C) most likely can be explained by "Ca2+ stabilizing" effects of Dimebon resulting from inhibition of NMDAR (Fig 3) and voltage-gated Ca2+ channels (Fig 4).
Ca2 Regulation (effects) of Gene_expression (channels) of Fig 4
1) Confidence 0.02 Published 2008 Journal Mol Neurodegener Section Body Doc Link PMC2577671 Disease Relevance 0.31 Pain Relevance 0.08

General Comments

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