INT251694

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Context Info
Confidence 0.06
First Reported 2007
Last Reported 2008
Negated 0
Speculated 1
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 0.05
Pain Relevance 0.52

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

pigmentation (Fig4) Golgi apparatus (Fig4) endoplasmic reticulum (Fig4)
Anatomy Link Frequency
brain 2
Fig4 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
agonist 21 100.00 Very High Very High Very High
Kinase C 78 98.48 Very High Very High Very High
Pyramidal cell 12 73.48 Quite High
cerebral cortex 16 30.68 Quite Low
tetrodotoxin 14 5.64 Low Low
Neurotransmitter 12 5.00 Very Low Very Low Very Low
Bile 8 5.00 Very Low Very Low Very Low
Glutamate 6 5.00 Very Low Very Low Very Low
nMDA receptor 4 5.00 Very Low Very Low Very Low
Glutamate receptor 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Urological Neuroanatomy 14 52.68 Quite High
Cirrhosis 38 5.00 Very Low Very Low Very Low
Hepatic Encephalopathy 34 5.00 Very Low Very Low Very Low
Cognitive Disorder 23 5.00 Very Low Very Low Very Low
Liver Failure 20 5.00 Very Low Very Low Very Low
Liver Disease 16 5.00 Very Low Very Low Very Low
Nash(non-alcoholic Steatohepatitis) 12 5.00 Very Low Very Low Very Low
Hyperammonemia 8 5.00 Very Low Very Low Very Low
Shock 4 5.00 Very Low Very Low Very Low
Dementia 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The levels of the T-transcript, the major transcript in mammalian brain that encodes subunits which produce monomeric and tetrameric forms, was unaltered in cortices from BDL rats compared to controls (Fig. 5A).
Regulation (unaltered) of Regulation (controls) of Fig in brain
1) Confidence 0.06 Published 2008 Journal Brain Section Body Doc Link PMC2577805 Disease Relevance 0.05 Pain Relevance 0.10
As expected for the unmodified ChAT enzyme activity, the levels of both cChAT and pChAT were unaltered in cortices from BDL rats compared to NL controls (Fig. 5A).
Regulation (unaltered) of Regulation (controls) of Fig
2) Confidence 0.06 Published 2008 Journal Brain Section Body Doc Link PMC2577805 Disease Relevance 0 Pain Relevance 0
translocation, nor did it appear to affect agonist-mediated M1 mACh receptor regulation (Fig. 8b).
Spec (appear) Regulation (affect) of Regulation (regulation) of Fig associated with agonist
3) Confidence 0.05 Published 2007 Journal Journal of Neurochemistry Section Body Doc Link PMC2658029 Disease Relevance 0 Pain Relevance 0.42

General Comments

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