INT252419

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Context Info
Confidence 0.08
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.21
Pain Relevance 0.16

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

generation of precursor metabolites and energy (Enpp1) extracellular space (Enpp1) plasma membrane (Enpp1)
Enpp1 (Mus musculus)
Pain Link Frequency Relevance Heat
adenocard 22 99.32 Very High Very High Very High
Glutamate 20 92.64 High High
long-term potentiation 2 47.36 Quite Low
depression 7 41.76 Quite Low
antiepileptic Drug 8 5.00 Very Low Very Low Very Low
Central nervous system 5 5.00 Very Low Very Low Very Low
Glutamate receptor 4 5.00 Very Low Very Low Very Low
Action potential 4 5.00 Very Low Very Low Very Low
nMDA receptor 3 5.00 Very Low Very Low Very Low
Neurotransmitter 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 1 90.80 High High
Epilepsy 29 64.60 Quite High
Convulsion 11 51.16 Quite High
Depression 7 41.76 Quite Low
Brain Injury 1 9.08 Low Low
Disease 4 5.00 Very Low Very Low Very Low
Injury 3 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low
Thrombosis 1 5.00 Very Low Very Low Very Low
Poisoning 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The mechanism for this is that ATP is rapidly broken down into adenosine by ectonucleotidases (E-NTPDase, E-NPP, alkaline phosphatises, and ecto-5?
Phosphorylation (phosphatises) of E-NPP associated with adenocard
1) Confidence 0.08 Published 2008 Journal Purinergic Signal Section Body Doc Link PMC2583203 Disease Relevance 0.21 Pain Relevance 0.16

General Comments

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