INT252720

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.06
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.82
Pain Relevance 0.05

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (IREB2) mitochondrion (IREB2) RNA binding (IREB2)
cytoplasm (IREB2)
Anatomy Link Frequency
macrophages 1
IREB2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 48 93.88 High High
cytokine 21 5.00 Very Low Very Low Very Low
fibrosis 19 5.00 Very Low Very Low Very Low
Inflammatory response 11 5.00 Very Low Very Low Very Low
chemokine 6 5.00 Very Low Very Low Very Low
alcohol 5 5.00 Very Low Very Low Very Low
Bile 3 5.00 Very Low Very Low Very Low
Paracetamol 2 5.00 Very Low Very Low Very Low
COX2 1 5.00 Very Low Very Low Very Low
Nerve growth factor 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Liver Disease 43 99.26 Very High Very High Very High
INFLAMMATION 57 93.88 High High
Stress 107 87.64 High High
Disease 18 75.04 Quite High
Iron Overload 5 54.56 Quite High
Wound Healing 12 50.00 Quite Low
Injury 46 45.60 Quite Low
Fibrosis 37 25.72 Quite Low
Alcoholic Liver Diseases 19 22.08 Low Low
Nash(non-alcoholic Steatohepatitis) 12 20.80 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
We do not know whether these regulatory mechanisms may be altered in CLDs, although it has been proposed that inactivation of IRP-2 (which is usually highly expressed in macrophages) by RNS may help to explain iron sequestration patterns of macrophages detected in tissues undergoing inflammation [125].
Negative_regulation (inactivation) of IRP-2 in macrophages associated with liver disease and inflammation
1) Confidence 0.06 Published 2008 Journal Fibrogenesis Tissue Repair Section Body Doc Link PMC2584013 Disease Relevance 0.82 Pain Relevance 0.05

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox