INT252760

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Context Info
Confidence 0.19
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.77
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (RIPK1) signal transduction (RIPK1) mitochondrion (RIPK1)
plasma membrane (RIPK1) cytoplasm (RIPK1)
RIPK1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 48 22.72 Low Low
cytokine 21 5.00 Very Low Very Low Very Low
fibrosis 19 5.00 Very Low Very Low Very Low
Inflammatory response 11 5.00 Very Low Very Low Very Low
chemokine 6 5.00 Very Low Very Low Very Low
alcohol 5 5.00 Very Low Very Low Very Low
Bile 3 5.00 Very Low Very Low Very Low
Paracetamol 2 5.00 Very Low Very Low Very Low
COX2 1 5.00 Very Low Very Low Very Low
Nerve growth factor 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Death 55 98.20 Very High Very High Very High
Apoptosis 55 65.48 Quite High
Stress 107 61.24 Quite High
Wound Healing 12 50.00 Quite Low
INFLAMMATION 57 22.72 Low Low
Liver Disease 43 20.68 Low Low
Injury 46 9.28 Low Low
Fatty Liver 24 8.48 Low Low
Cirrhosis 45 5.00 Very Low Very Low Very Low
Fibrosis 37 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
ROS may even operate by inducing a PARP-1 over-activation resulting in depletion of NAD+ and ATP as well as in a downstream involvement of TRAF2/RIP1 mediating sustained JNK activation and cell death, but it is still unclear how TRAF2 and RIP1 may be involved following PARP-1 activation [207,208].
RIP1 Binding (involvement) of associated with death
1) Confidence 0.19 Published 2008 Journal Fibrogenesis Tissue Repair Section Body Doc Link PMC2584013 Disease Relevance 0.77 Pain Relevance 0

General Comments

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