INT253761

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Context Info
Confidence 0.03
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.73
Pain Relevance 0.12

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Hmgb1, Gopc) transport (Gopc) cell morphogenesis (Hmgb1)
Golgi apparatus (Gopc) DNA binding (Hmgb1) extracellular space (Hmgb1)
Anatomy Link Frequency
monocytes 2
Hmgb1 (Mus musculus)
Gopc (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammatory response 27 96.44 Very High Very High Very High
chemokine 5 86.96 High High
Inflammatory stimuli 2 59.96 Quite High
Inflammation 36 9.28 Low Low
cytokine 23 8.24 Low Low
Inflammatory mediators 15 5.00 Very Low Very Low Very Low
vagus nerve 6 5.00 Very Low Very Low Very Low
cINOD 5 5.00 Very Low Very Low Very Low
Neuropeptide 3 5.00 Very Low Very Low Very Low
rheumatoid arthritis 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cancer 5 100.00 Very High Very High Very High
INFLAMMATION 83 96.08 Very High Very High Very High
Infection 18 92.24 High High
Dengue 1 92.08 High High
Anaemia 1 91.36 High High
Injury 14 91.20 High High
Necrosis 4 52.80 Quite High
Apoptosis 11 37.60 Quite Low
Adhesions 1 6.16 Low Low
Sepsis 99 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Accumulating evidence indicates that HMGB1 can stimulating migration of neurites (Ref.
             61), smooth muscle cells (Ref. 62), tumour cells (Ref. 63), mesoangioblast stem cells (Refs 64, 65), monocytes (Ref.
             66), dendritic cells (Refs 67, 68)
           and neutrophils (Ref. 69) (Fig. 2). 
HMGB1 Positive_regulation (stimulating) of Gene_expression (migration) of Fig in monocytes associated with cancer
1) Confidence 0.03 Published 2008 Journal Expert Reviews in Molecular Medicine Section Body Doc Link PMC2586610 Disease Relevance 0.73 Pain Relevance 0.12

General Comments

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