INT254459

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Context Info
Confidence 0.03
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Ca2, Calm2) cytosol (Ca2, Calm2) nucleoplasm (Calm2)
lyase activity (Ca2) response to stress (Ca2) extracellular space (Ca2)
Ca2 (Rattus norvegicus)
Calm2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
addiction 17 98.08 Very High Very High Very High
tetrodotoxin 5 5.00 Very Low Very Low Very Low
imagery 4 5.00 Very Low Very Low Very Low
Action potential 3 5.00 Very Low Very Low Very Low
Nerve growth factor 1 5.00 Very Low Very Low Very Low
Neurotransmitter 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 14 5.00 Very Low Very Low Very Low
Myelodysplastic Syndromes 3 5.00 Very Low Very Low Very Low
Infection 3 5.00 Very Low Very Low Very Low
Targeted Disruption 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Even when Ca2+ influx is sharply reduced (e.g., by Cd2+ block), the local Ca2+ rise would still be sufficient to activate most of the local CaM molecules (Fig. 8 A, right), hence the shallow dependence on the open channel Ca2+ flux (Fig. 8 B, right).
Ca2 Positive_regulation (activate) of CaM associated with addiction
1) Confidence 0.03 Published 2008 Journal The Journal of Cell Biology Section Body Doc Link PMC2592819 Disease Relevance 0 Pain Relevance 0.08

General Comments

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