INT254498

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Context Info
Confidence 0.05
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.34
Pain Relevance 0.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (SREBF1, GOPC) nuclear envelope (SREBF1) endoplasmic reticulum (SREBF1)
DNA binding (SREBF1) cytoplasm (GOPC) lipid metabolic process (SREBF1)
Anatomy Link Frequency
adipose tissue 1
SREBF1 (Homo sapiens)
GOPC (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 14 59.16 Quite High
antagonist 2 56.40 Quite High
Inflammatory response 2 5.00 Very Low Very Low Very Low
lidocaine 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Inflammatory marker 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Obesity 80 99.44 Very High Very High Very High
INFLAMMATION 17 59.16 Quite High
Overweight 79 50.00 Quite Low
Stress 3 43.24 Quite Low
Adhesions 6 5.00 Very Low Very Low Very Low
Apoptosis 2 5.00 Very Low Very Low Very Low
Insulin Resistance 1 5.00 Very Low Very Low Very Low
Weight Loss 1 5.00 Very Low Very Low Very Low
Metabolic Syndrome 1 5.00 Very Low Very Low Very Low
Disease 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This implicates that, in addition to liver, HNF4-alpha could be an important regulator of gene expression and energy metabolism in adipose tissue, at least in lean subjects.Fig. 5Network of direct interactions between transcription factors HNF4alpha, SREBP1 and PPARA and genes involved in energy metabolism that were differentially expressed in lean subjects in response to intervention spread compared to control spread.
SREBP1 Binding (interactions) of Fig in adipose tissue associated with obesity
1) Confidence 0.05 Published 2008 Journal Genes Nutr Section Body Doc Link PMC2593008 Disease Relevance 0.34 Pain Relevance 0.08

General Comments

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