INT254631

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.01
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 3
Disease Relevance 2.79
Pain Relevance 1.51

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (Lamb1) extracellular region (Lamb1) cell adhesion (Lamb1)
proteinaceous extracellular matrix (Lamb1) enzyme binding (Lamb1)
Lamb1 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 72 99.66 Very High Very High Very High
b2 receptor 51 98.96 Very High Very High Very High
B1 receptor 9 98.54 Very High Very High Very High
Pain 3 97.00 Very High Very High Very High
cytokine 24 92.36 High High
Central nervous system 57 92.16 High High
bradykinin 36 91.00 High High
Multiple sclerosis 42 88.00 High High
antagonist 15 86.12 High High
chemokine 60 78.48 Quite High
Disease Link Frequency Relevance Heat
INFLAMMATION 84 99.66 Very High Very High Very High
Pain 3 97.00 Very High Very High Very High
Adhesions 39 91.64 High High
Multiple Sclerosis 258 88.00 High High
Disease 45 76.60 Quite High
Shock 3 76.40 Quite High
Brain Injury 3 73.36 Quite High
Injury 3 39.36 Quite Low
Targeted Disruption 12 35.76 Quite Low
Recurrence 3 32.08 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
B2 is partially necessary for B1 receptor induction as supported by the consistent finding of B2 participation in B1 mRNA and protein induction in several models of inflammation [60].
Positive_regulation (induction) of B1 associated with inflammation and b1 receptor
1) Confidence 0.01 Published 2008 Journal J Neuroinflammation Section Body Doc Link PMC2596102 Disease Relevance 1.06 Pain Relevance 0.49
The actions of kinins are mediated through stimulation of two subtypes of seven-transmembrane-domain G-protein-coupled receptors, namely B1 and B2.
Positive_regulation (stimulation) of B1
2) Confidence 0.00 Published 2008 Journal J Neuroinflammation Section Body Doc Link PMC2596102 Disease Relevance 0.85 Pain Relevance 0.58
Kinins are important mediators of inflammation and act through stimulation of two receptor subtypes, B1 and B2.
Positive_regulation (stimulation) of B1 associated with inflammation
3) Confidence 0.00 Published 2008 Journal J Neuroinflammation Section Abstract Doc Link PMC2596102 Disease Relevance 0.88 Pain Relevance 0.44

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox