INT254639

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.18
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.51
Pain Relevance 0.08

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

small molecule metabolic process (CGA, AGT) extracellular region (CGA, AGT) cell-cell signaling (CGA, AGT)
extracellular space (AGT) signal transduction (CGA) aging (AGT)
CGA (Homo sapiens)
AGT (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 1 98.24 Very High Very High Very High
iatrogenic 1 65.84 Quite High
abdominal pain 2 11.52 Low Low
cva 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Edema 3 99.44 Very High Very High Very High
Necrosis 1 99.02 Very High Very High Very High
Cancer 1 98.84 Very High Very High Very High
Ovarian Hyperstimulation Syndrome 29 96.56 Very High Very High Very High
Syndrome 2 83.72 Quite High
Pressure And Volume Under Development 12 65.20 Quite High
Pleural Effusion 14 64.48 Quite High
Dyspnea 5 57.20 Quite High
Body Weight 1 37.84 Quite Low
Volume Depletion And Dehydration 1 23.36 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
There is increasing evidence that certain vasoactive substances such as vascular endothelial growth factor (VEGF), cytokines (IL-2, IL-6, and IL-8), tumor necrosis factor-alpha (TNF-alpha), and the ovarian renin-angiotensin system, which are activated by gonadotropin, can lead to increased vascular permeability and extravascular fluid accumulation in OHSS [6,7].
gonadotropin Positive_regulation (activated) of Gene_expression (system) of angiotensin associated with ovarian hyperstimulation syndrome, necrosis, cancer, edema and cytokine
1) Confidence 0.18 Published 2008 Journal Cases J Section Body Doc Link PMC2596110 Disease Relevance 1.51 Pain Relevance 0.08

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox