INT254741

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Context Info
Confidence 0.20
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Skil) DNA binding (Skil) molecular_function (Skil)
cytoplasm (Skil)
Skil (Rattus norvegicus)
Pain Link Frequency Relevance Heat
ischemia 20 5.00 Very Low Very Low Very Low
anesthesia 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cv Unclassified Under Development 18 5.00 Very Low Very Low Very Low
Apoptosis 14 5.00 Very Low Very Low Very Low
Injury 10 5.00 Very Low Very Low Very Low
Aids-related Complex 4 5.00 Very Low Very Low Very Low
Reperfusion Injury 4 5.00 Very Low Very Low Very Low
Diabetes Mellitus 2 5.00 Very Low Very Low Very Low
Coronary Artery Disease 2 5.00 Very Low Very Low Very Low
Bacterial Respiratory Disease 2 5.00 Very Low Very Low Very Low
Death 2 5.00 Very Low Very Low Very Low
Hepatocellular Cancer 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
As shown in
Fig. 6, A and
B, the decrease in SNO-HSA caused by HepG2 cells occurred
more rapidly for SNO-HSA-hd (+) than SNO-HSA-hd (-). 
Positive_regulation (occurred) of Negative_regulation (decrease) of SNO-HSA
1) Confidence 0.20 Published 2008 Journal The Journal of Biological Chemistry Section Body Doc Link PMC2596408 Disease Relevance 0 Pain Relevance 0
From Fig.
5B, it is apparent that the decrease in SNO-HSA caused by
this cell type was faster and quantitatively more pronounced in the presence
of OA, and that the effects increased with OA concentration. 
Positive_regulation (caused) of Negative_regulation (decrease) of SNO-HSA
2) Confidence 0.20 Published 2008 Journal The Journal of Biological Chemistry Section Body Doc Link PMC2596408 Disease Relevance 0 Pain Relevance 0

General Comments

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