INT254883

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Context Info
Confidence 0.05
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.14
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (Htt) endoplasmic reticulum (Htt) mitochondrion organization (Htt)
embryo development (Htt) protein complex (Htt) cytoplasm (Htt)
Htt (Mus musculus)
Pain Link Frequency Relevance Heat
addiction 1 5.00 Very Low Very Low Very Low
agonist 1 5.00 Very Low Very Low Very Low
Clonidine 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Calcium channel 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 45 99.44 Very High Very High Very High
Frailty 1 79.24 Quite High
Spinocerebellar Ataxia Type 2 1 74.16 Quite High
Tauopathy 2 69.84 Quite High
Neurodegenerative Disease 12 65.92 Quite High
Multiple Sulfatase Deficiency Disease 9 51.64 Quite High
Targeted Disruption 15 18.96 Low Low
Motor Neuron Diseases 15 5.00 Very Low Very Low Very Low
Starvation 11 5.00 Very Low Very Low Very Low
Death 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Inhibition of autophagy by chemical regulators, such as bafilomycin A1 and 3-methyladenine, decreases the clearance of aggregate-prone proteins such as mutant huntingtin (that causes Huntington's disease) or mutant forms of alpha-synuclein that cause familial Parkinson's disease.
Negative_regulation (decreases) of Positive_regulation (causes) of mutant huntingtin associated with disease
1) Confidence 0.05 Published 2008 Journal Biochim Biophys Acta Section Body Doc Link PMC2597715 Disease Relevance 1.14 Pain Relevance 0

General Comments

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