INT255380

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Context Info
Confidence 0.02
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0
Pain Relevance 1.28

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

AIS (Homo sapiens)
Nav1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Nav1.2 176 100.00 Very High Very High Very High
sodium channel 124 86.56 High High
potassium channel 14 75.52 Quite High
Glutamate 12 57.64 Quite High
Hippocampus 8 5.04 Very Low Very Low Very Low
Peripheral nervous system 6 5.00 Very Low Very Low Very Low
Nav1.6 4 5.00 Very Low Very Low Very Low
Action potential 4 5.00 Very Low Very Low Very Low
Sciatic nerve 4 5.00 Very Low Very Low Very Low
Central nervous system 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 2 34.72 Quite Low
Adhesions 8 5.00 Very Low Very Low Very Low
Sprains And Strains 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The fact that the association of E1111A with either S1124 or S1126 mutations was mandatory for the loss of Kv2.1-Nav1.2 segregation at the AIS was consistent with this conclusion.
AIS Binding (segregation) of Nav1 associated with nav1.2
1) Confidence 0.02 Published 2008 Journal The Journal of Cell Biology Section Body Doc Link PMC2600743 Disease Relevance 0 Pain Relevance 0.60
However, this mutation did not impair Kv2.1-Nav1.2 segregation at the AIS because of the contribution of the phosphoserine residues.
AIS Binding (segregation) of Nav1 associated with nav1.2
2) Confidence 0.01 Published 2008 Journal The Journal of Cell Biology Section Body Doc Link PMC2600743 Disease Relevance 0 Pain Relevance 0.68

General Comments

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