INT255432

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Context Info
Confidence 0.35
First Reported 2008
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 1.41
Pain Relevance 0.16

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (SAG)
Anatomy Link Frequency
brain 1
SAG (Homo sapiens)
Pain Link Frequency Relevance Heat
headache 3 87.16 High High
cytokine 32 78.28 Quite High
TRP channel 7 5.00 Very Low Very Low Very Low
abdominal pain 3 5.00 Very Low Very Low Very Low
agonist 3 5.00 Very Low Very Low Very Low
Inflammation 2 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low
Central nervous system 1 5.00 Very Low Very Low Very Low
Neuropathic pain 1 5.00 Very Low Very Low Very Low
Peripheral nervous system 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Brain Abscess 11 99.12 Very High Very High Very High
Abscess 21 93.92 High High
Fever 9 87.44 High High
Headache 3 87.16 High High
Leishmaniasis 33 84.36 Quite High
Disease 14 79.52 Quite High
Head Trauma 1 74.20 Quite High
Infection 14 72.04 Quite High
Cv Unclassified Under Development 14 67.60 Quite High
Liver Abscess 7 56.24 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
-Arrestin 1 interacts with, and acts as an adaptor for AIP4, an E3 ubiquitin ligase responsible for TRPV4 ubiquitination.
Arrestin 1 Binding (interacts) of
1) Confidence 0.35 Published 2010 Journal The Journal of Biological Chemistry Section Abstract Doc Link PMC2943294 Disease Relevance 0 Pain Relevance 0
The SAG has been recently recognized as a common cause of brain abscesses, which were collectively identified with other streptococci as viridans group streptococci in prior studies [29,31].
SAG Binding (recognized) of in brain associated with brain abscess
2) Confidence 0.22 Published 2008 Journal BMC Infect Dis Section Body Doc Link PMC2600825 Disease Relevance 1.06 Pain Relevance 0.09
In addition to the direct antileishmanial activity, both AmB and SAG showed in vitro immunomodulatory activities on PBMCs resulting in decline of IL-10, with AmB downregulating TGF-?
SAG Binding (showed) of
3) Confidence 0.05 Published 2010 Journal PLoS Neglected Tropical Diseases Section Body Doc Link PMC2910702 Disease Relevance 0.35 Pain Relevance 0.08

General Comments

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