INT257741

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Context Info
Confidence 0.15
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.06
Pain Relevance 0.35

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (HRAS, PTEN) cytosol (HRAS, PTEN) nucleus (HRAS, PTEN)
plasma membrane (HRAS, PTEN) Golgi apparatus (HRAS) enzyme binding (PTEN)
HRAS (Homo sapiens)
PTEN (Homo sapiens)
Pain Link Frequency Relevance Heat
endometriosis 108 99.14 Very High Very High Very High
pain pelvic 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
Inflammation 1 5.00 Very Low Very Low Very Low
Thalamus 1 5.00 Very Low Very Low Very Low
Dismenorea 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Endometriosis 138 99.14 Very High Very High Very High
Disease 11 98.92 Very High Very High Very High
Ovarian Cancer 15 65.60 Quite High
Reprotox - General 3 4 5.00 Very Low Very Low Very Low
Cancer 3 5.00 Very Low Very Low Very Low
Carcinoma 3 5.00 Very Low Very Low Very Low
Myelodysplastic Syndromes 2 5.00 Very Low Very Low Very Low
Malignant Neoplastic Disease 2 5.00 Very Low Very Low Very Low
Reprotox - General 1 1 5.00 Very Low Very Low Very Low
Gynecological Disease 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
As overexpression of AXL and SHC1 theoretically activates both PI3K-Akt and MAPK pathways, dysregulation of these genes in human endometriosis may correspond to oncogenic activation of K-ras or biallelic losses of Pten in the mouse model of the disease.
K-ras Negative_regulation (losses) of Pten associated with endometriosis and disease
1) Confidence 0.15 Published 2008 Journal Reprod Biol Endocrinol Section Body Doc Link PMC2615013 Disease Relevance 1.06 Pain Relevance 0.35

General Comments

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