INT257876

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Context Info
Confidence 0.03
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.63
Pain Relevance 0.03

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell death (Rraga) Golgi apparatus (Rraga) nucleus (Rraga)
cellular_component (Rraga) biological_process (Rraga) lysosome (Rraga)
Rraga (Rattus norvegicus)
Pik3cg (Rattus norvegicus)
Pain Link Frequency Relevance Heat
withdrawal 1 51.88 Quite High
Inflammation 3 29.68 Quite Low
agonist 3 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
ketamine 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
imagery 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Neointima 11 76.20 Quite High
Injury 16 74.60 Quite High
Vasculitis 2 53.96 Quite High
Hyperglycemia 2 43.04 Quite Low
Insulin Resistance 2 40.64 Quite Low
Diabetes Mellitus 7 38.32 Quite Low
INFLAMMATION 3 29.68 Quite Low
Hyperplasia 4 18.64 Low Low
Retinoblastoma 2 5.00 Very Low Very Low Very Low
Starvation 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
PI3K and MAPK are upstream molecules and related to the insulin-mediated signaling pathway, and insulin can activate PI3K through the phosphorylation of IRS1/2 or MAPK by Ras-GTP.
Ras-GTP Positive_regulation (activate) of PI3K
1) Confidence 0.03 Published 2008 Journal Yonsei Medical Journal Section Body Doc Link PMC2615285 Disease Relevance 0.63 Pain Relevance 0.03

General Comments

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