INT258890

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Context Info
Confidence 0.13
First Reported 2007
Last Reported 2007
Negated 1
Speculated 0
Reported most in Body
Documents 1
Total Number 3
Disease Relevance 0.71
Pain Relevance 0.23

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
hearts 6
Shbdp1 (Mus musculus)
Pain Link Frequency Relevance Heat
Action potential 69 85.24 High High
lidocaine 102 46.68 Quite Low
addiction 6 33.28 Quite Low
action potential duration 45 5.00 Very Low Very Low Very Low
antiarrhythmic agent 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Bartter Syndrome 45 93.68 High High
Hypokalemia 6 88.40 High High
Syndrome 30 63.12 Quite High
Heart Rate Under Development 18 5.00 Very Low Very Low Very Low
Arrhythmogenic Right Ventricular Dysplasia 9 5.00 Very Low Very Low Very Low
Coronary Heart Disease 6 5.00 Very Low Very Low Very Low
Heart Arrhythmia 6 5.00 Very Low Very Low Very Low
Heart Disease 3 5.00 Very Low Very Low Very Low
Aneurism 3 5.00 Very Low Very Low Very Low
Dislocations 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
While studies under normokalaemic conditions confirmed an absence of arrhythmic activity at either BCL (0 out of five hearts subjected to regular stimulation, 0 out of seven hearts subjected to S2 stimulation), studies under hypokalaemic conditions (Fig. 1) revealed arrhythmogenicity at the longest (A, five out of five hearts subjected to regular stimulation, seven out of seven hearts subjected to S2 stimulation) but not the shortest (B, 0 out of five hearts subjected to regular stimulation, one out of nine hearts subjected to S2 stimulation) BCL studied.
Neg (not) Positive_regulation (subjected) of Positive_regulation (stimulation) of S2 in hearts
1) Confidence 0.13 Published 2007 Journal Pflugers Arch Section Body Doc Link PMC2627988 Disease Relevance 0.25 Pain Relevance 0.08
While studies under normokalaemic conditions confirmed an absence of arrhythmic activity at either BCL (0 out of five hearts subjected to regular stimulation, 0 out of seven hearts subjected to S2 stimulation), studies under hypokalaemic conditions (Fig. 1) revealed arrhythmogenicity at the longest (A, five out of five hearts subjected to regular stimulation, seven out of seven hearts subjected to S2 stimulation) but not the shortest (B, 0 out of five hearts subjected to regular stimulation, one out of nine hearts subjected to S2 stimulation) BCL studied.
Positive_regulation (subjected) of Positive_regulation (stimulation) of S2 in hearts
2) Confidence 0.13 Published 2007 Journal Pflugers Arch Section Body Doc Link PMC2627988 Disease Relevance 0.19 Pain Relevance 0.07
While studies under normokalaemic conditions confirmed an absence of arrhythmic activity at either BCL (0 out of five hearts subjected to regular stimulation, 0 out of seven hearts subjected to S2 stimulation), studies under hypokalaemic conditions (Fig. 1) revealed arrhythmogenicity at the longest (A, five out of five hearts subjected to regular stimulation, seven out of seven hearts subjected to S2 stimulation) but not the shortest (B, 0 out of five hearts subjected to regular stimulation, one out of nine hearts subjected to S2 stimulation) BCL studied.
Positive_regulation (subjected) of Positive_regulation (stimulation) of S2 in hearts
3) Confidence 0.13 Published 2007 Journal Pflugers Arch Section Body Doc Link PMC2627988 Disease Relevance 0.27 Pain Relevance 0.08

General Comments

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