INT259011

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Context Info
Confidence 0.25
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.25
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (TNFSF18) extracellular space (TNFSF18) plasma membrane (TNFSF18)
cell-cell signaling (TNFSF18)
TNFSF18 (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 8 48.24 Quite Low
Inflammation 6 45.84 Quite Low
Sciatica 6 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
backache 2 5.00 Very Low Very Low Very Low
Cauda equina syndrome 2 5.00 Very Low Very Low Very Low
metalloproteinase 2 5.00 Very Low Very Low Very Low
imagery 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Intervertebral Disk Displacement 18 98.16 Very High Very High Very High
Autoimmune Disease 20 59.12 Quite High
Necrosis 10 50.00 Quite Low
Cancer 10 50.00 Quite Low
INFLAMMATION 8 45.84 Quite Low
Spinal Fractures 8 5.00 Very Low Very Low Very Low
Nerve Root Compression 6 5.00 Very Low Very Low Very Low
Polyradiculopathy 2 5.00 Very Low Very Low Very Low
Low Back Pain 2 5.00 Very Low Very Low Very Low
Injury 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The mRNA expression of AITR and AITRL were determined by reverse transcriptase-polymerase chain reaction (RT-PCR).
Transcription (expression) of AITRL
1) Confidence 0.25 Published 2007 Journal Yonsei Medical Journal Section Body Doc Link PMC2628152 Disease Relevance 0 Pain Relevance 0
The expression of AITRL mRNA was in creased in the patients when compared to the controls, but there was no difference in AITR mRNA expression between the two groups (Fig. 7).
Transcription (expression) of AITRL
2) Confidence 0.21 Published 2007 Journal Yonsei Medical Journal Section Body Doc Link PMC2628152 Disease Relevance 0.25 Pain Relevance 0

General Comments

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