INT259768

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Context Info
Confidence 0.00
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.32
Pain Relevance 0.62

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Agtr1a, Etf1) endosome (Agtr1a) aging (Agtr1a)
Golgi apparatus (Agtr1a) plasma membrane (Agtr1a) signal transducer activity (Agtr1a)
Agtr1a (Rattus norvegicus)
Etf1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
dorsal root ganglion 71 97.58 Very High Very High Very High
agonist 3 86.48 High High
antagonist 2 79.84 Quite High
adenocard 22 74.28 Quite High
Calcium channel 1 23.84 Low Low
Sciatic nerve 15 5.00 Very Low Very Low Very Low
withdrawal 8 5.00 Very Low Very Low Very Low
hyperexcitability 6 5.00 Very Low Very Low Very Low
Neuropathic pain 5 5.00 Very Low Very Low Very Low
Spinal cord 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Ganglion Cysts 72 97.58 Very High Very High Very High
Nerve Compression Syndromes 45 92.48 High High
Neuropathic Pain 19 85.12 High High
Pain 8 5.00 Very Low Very Low Very Low
Nociception 7 5.00 Very Low Very Low Very Low
Injury 7 5.00 Very Low Very Low Very Low
Nervous System Injury 4 5.00 Very Low Very Low Very Low
Peripheral Neuropathy 3 5.00 Very Low Very Low Very Low
INFLAMMATION 2 5.00 Very Low Very Low Very Low
Hyperalgesia 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
However, in the continuous presence of NCPA (n = 7 DRG, 4 rats), there was a significant decrease in both basal and KCl-evoked ATP release (Fig. 3A, B), suggesting functional modulation of ATP release by A1R activation.


Positive_regulation (activation) of A1R Regulation (modulation) of release associated with dorsal root ganglion
1) Confidence 0.00 Published 2008 Journal Mol Pain Section Body Doc Link PMC2630978 Disease Relevance 1.32 Pain Relevance 0.62

General Comments

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