INT259848

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Context Info
Confidence 0.12
First Reported 2008
Last Reported 2010
Negated 0
Speculated 1
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 2.10
Pain Relevance 0.30

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Slc12a3) transport (Slc12a3) plasma membrane (Slc12a3)
transmembrane transport (Slc12a3)
Anatomy Link Frequency
neural crest 1
brains 1
Slc12a3 (Mus musculus)
Pain Link Frequency Relevance Heat
Central nervous system 32 95.04 Very High Very High Very High
ischemia 1 71.80 Quite High
Inflammation 6 5.00 Very Low Very Low Very Low
cytokine 3 5.00 Very Low Very Low Very Low
anesthesia 3 5.00 Very Low Very Low Very Low
Inflammatory response 3 5.00 Very Low Very Low Very Low
imagery 3 5.00 Very Low Very Low Very Low
midbrain 2 5.00 Very Low Very Low Very Low
Pyramidal cell 2 5.00 Very Low Very Low Very Low
Hippocampus 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Neurocysticercosis 23 100.00 Very High Very High Very High
Targeted Disruption 27 98.48 Very High Very High Very High
Infection 31 95.84 Very High Very High Very High
Central Nervous System Disease 28 95.04 Very High Very High Very High
Cleft Palate 8 86.84 High High
Dwarfism 1 76.44 Quite High
Autoimmune Disease 2 73.12 Quite High
Cv Unclassified Under Development 1 71.80 Quite High
Polydactyly 1 71.32 Quite High
Ventricular Heart Septal Defects 4 69.12 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
To investigate the role of Spry1 in regulating NCC during development, we induced tissue-specific expression of Spry1 using Cre/loxP recombination in the neural crest lineage by using Wnt1-Cre transgenic mice [20].
Spec (investigate) Regulation (regulating) of NCC in neural crest associated with targeted disruption
1) Confidence 0.12 Published 2010 Journal BMC Dev Biol Section Body Doc Link PMC2874773 Disease Relevance 0.80 Pain Relevance 0
This study and our previous findings [12] indicate that TLRs 1–13 exhibit differential expression and regulation in normal and NCC mouse brains.
Regulation (regulation) of NCC in brains associated with neurocysticercosis
2) Confidence 0.08 Published 2008 Journal J Neuroinflammation Section Body Doc Link PMC2631477 Disease Relevance 1.30 Pain Relevance 0.30

General Comments

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