INT25987

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Context Info
Confidence 0.06
First Reported 1989
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 3
Disease Relevance 0.43
Pain Relevance 1.59

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Vipr1) plasma membrane (Vipr1) nucleus (Vipr1)
cytoplasm (Vipr1) signal transducer activity (Vipr1)
Anatomy Link Frequency
sensory neurons 1
cerebral cortex 1
Vipr1 (Mus musculus)
Pain Link Frequency Relevance Heat
calcitonin gene related peptide 14 100.00 Very High Very High Very High
substance P 12 100.00 Very High Very High Very High
bradykinin 6 100.00 Very High Very High Very High
Neuropeptide 2 100.00 Very High Very High Very High
cerebral cortex 3 99.60 Very High Very High Very High
Neurotransmitter 7 98.96 Very High Very High Very High
antagonist 37 97.84 Very High Very High Very High
Somatostatin 4 84.56 Quite High
agonist 13 82.56 Quite High
tetrodotoxin 4 79.48 Quite High
Disease Link Frequency Relevance Heat
Nociception 16 97.94 Very High Very High Very High
INFLAMMATION 21 69.28 Quite High
Hyperalgesia 7 68.24 Quite High
Pain 38 5.00 Very Low Very Low Very Low
Targeted Disruption 12 5.00 Very Low Very Low Very Low
Diabetes Mellitus 6 5.00 Very Low Very Low Very Low
Overactive Bladder 6 5.00 Very Low Very Low Very Low
Neuropathic Pain 6 5.00 Very Low Very Low Very Low
Increased Venous Pressure Under Development 6 5.00 Very Low Very Low Very Low
Anxiety Disorder 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Release of vasoactive intestinal peptide in mouse cerebral cortex: evidence for a role of arachidonic acid metabolites.
Localization (Release) of intestinal peptide in cerebral cortex associated with cerebral cortex
1) Confidence 0.06 Published 1989 Journal J. Neurosci. Section Title Doc Link 2545840 Disease Relevance 0 Pain Relevance 0.39
Descending relaxation was accompanied by vasoactive intestinal peptide (VIP) release, a marker for inhibitory neurotransmitter release, into the caudad compartment, and was partly inhibited by VIP10-28 and NG-nitro-L-arginine, and abolished by a combination of the two agents.
Localization (release) of intestinal peptide associated with neurotransmitter
2) Confidence 0.04 Published 2003 Journal J. Pharmacol. Exp. Ther. Section Abstract Doc Link 12966154 Disease Relevance 0 Pain Relevance 0.58
Activation of Ca2+ permeant nociceptive ion channels on the peripheral and central terminals of sensory neurons leads to the synthesis and/or release of a variety of proinflammatory agents and neuropeptides, like ATP, BK, PGs, CGRP, SP and vasoactive intestinal peptide (VIP) [105, 114].
Localization (release) of intestinal peptide in sensory neurons associated with nociception, calcitonin gene related peptide, neuropeptide, bradykinin and substance p
3) Confidence 0.01 Published 2008 Journal Current Neuropharmacology Section Body Doc Link PMC2647151 Disease Relevance 0.43 Pain Relevance 0.62

General Comments

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