INT260738

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Context Info
Confidence 0.02
First Reported 2008
Last Reported 2009
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 2.22
Pain Relevance 0.50

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transferase activity, transferring glycosyl groups (Ugt1a2) endoplasmic reticulum (Ugt1a2) enzyme binding (Ugt1a2)
Ugt1a2 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammatory response 58 98.24 Very High Very High Very High
Inflammation 102 97.68 Very High Very High Very High
cytokine 16 85.36 High High
Inflammatory mediators 3 76.60 Quite High
Bile 58 47.80 Quite Low
ischemia 24 5.00 Very Low Very Low Very Low
fibrosis 16 5.00 Very Low Very Low Very Low
metalloproteinase 7 5.00 Very Low Very Low Very Low
chemokine 6 5.00 Very Low Very Low Very Low
Pain 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cholestasis 33 99.92 Very High Very High Very High
INFLAMMATION 158 97.88 Very High Very High Very High
Jaundice 8 95.64 Very High Very High Very High
Stress 34 95.36 Very High Very High Very High
Infection 9 82.88 Quite High
Injury 59 82.00 Quite High
Hypoxia 19 81.40 Quite High
Endotoxemia 2 69.28 Quite High
Obstructive Jaundice 6 58.56 Quite High
Sepsis 14 54.48 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Bilirubin is produced via reduction of heme-derived biliverdin by biliverdin reductase [110].
Gene_expression (produced) of Bilirubin
1) Confidence 0.02 Published 2008 Journal Fibrogenesis Tissue Repair Section Body Doc Link PMC2637833 Disease Relevance 1.26 Pain Relevance 0.25
Therefore, the increase in the production of bilirubin in the bruised tissue may have beneficiary effects as an inflammatory modulator.
Gene_expression (production) of bilirubin associated with inflammation
2) Confidence 0.01 Published 2009 Journal J Transl Med Section Body Doc Link PMC2667492 Disease Relevance 0.97 Pain Relevance 0.25

General Comments

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