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Context Info
Confidence 0.05
First Reported 2007
Last Reported 2007
Negated 0
Speculated 1
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.12
Pain Relevance 0.56

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Prkaca, Ephb1) plasma membrane (Prkaca, Ephb1) endosome (Ephb1)
nucleoplasm (Prkaca) mitochondrion (Prkaca) Golgi apparatus (Prkaca)
Prkaca (Rattus norvegicus)
Ephb1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Kinase C 3 100.00 Very High Very High Very High
substance P 5 92.40 High High
Spinal cord 22 81.60 Quite High
Glutamate receptor 1 79.24 Quite High
intrathecal 7 76.88 Quite High
antagonist 1 73.52 Quite High
Pain 24 66.84 Quite High
Glutamate 4 63.96 Quite High
bradykinin 1 52.20 Quite High
Inflammation 20 49.76 Quite Low
Disease Link Frequency Relevance Heat
Pain 25 66.84 Quite High
Spinal Cord Injury 8 56.16 Quite High
INFLAMMATION 16 49.76 Quite Low
Neuropathic Pain 16 35.80 Quite Low
Arthralgia 2 28.16 Quite Low
Nociception 8 5.00 Very Low Very Low Very Low
Injury 6 5.00 Very Low Very Low Very Low
Hyperalgesia 5 5.00 Very Low Very Low Very Low
Arthritis 4 5.00 Very Low Very Low Very Low
Apoptosis 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Finally, the contribution of other signalling pathways should not be ruled out as it has been verified that activation of the PKC and PKA pathways may lead to ERK phosphorylation [54, 108].
Positive_regulation (activation) of PKA Spec (may) Positive_regulation (lead) of Phosphorylation (phosphorylation) of ERK associated with kinase c
1) Confidence 0.05 Published 2007 Journal Current Neuropharmacology Section Body Doc Link PMC2644492 Disease Relevance 0.12 Pain Relevance 0.56

General Comments

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