INT261512

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.02
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.38
Pain Relevance 0.22

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (GPLD1) proteinaceous extracellular matrix (GPLD1)
GPLD1 (Homo sapiens)
Pain Link Frequency Relevance Heat
addiction 15 67.40 Quite High
Cannabinoid 30 64.16 Quite High
Multiple sclerosis 1 63.60 Quite High
Pain 1 61.24 Quite High
Dopamine 83 50.00 Quite Low
Endocannabinoid 14 50.00 Quite Low
midbrain 7 50.00 Quite Low
Neurotransmitter 3 24.20 Low Low
Ventral tegmentum 16 5.00 Very Low Very Low Very Low
Glutamate 12 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Drug Dependence 11 67.40 Quite High
Anxiety Disorder 2 65.16 Quite High
Demyelinating Disease 1 63.60 Quite High
Neurological Disease 3 62.64 Quite High
Obesity 2 61.56 Quite High
Pain 1 61.24 Quite High
Disease 31 5.00 Very Low Very Low Very Low
Schizophrenia 6 5.00 Very Low Very Low Very Low
Depression 6 5.00 Very Low Very Low Very Low
Injury 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
On the other hand, 2-AG derives from the metabolism of triacylglycerols, through receptor-dependent activation of phosphatidylinositol-specific phospholipase A1 (PLA1) and/or C (PLC) [196].
Positive_regulation (activation) of phosphatidylinositol-specific phospholipase A1
1) Confidence 0.02 Published 2007 Journal Current Neuropharmacology Section Body Doc Link PMC2644494 Disease Relevance 0.38 Pain Relevance 0.22

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox