INT261770

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.01
First Reported 2008
Last Reported 2008
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.06

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular region (Clcf1)
Clcf1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
gABA 36 77.84 Quite High
Calcium channel 4 51.04 Quite High
GABAergic 5 6.76 Low Low
Hippocampus 11 5.00 Very Low Very Low Very Low
bDMF 10 5.00 Very Low Very Low Very Low
agonist 6 5.00 Very Low Very Low Very Low
anticonvulsant 5 5.00 Very Low Very Low Very Low
antagonist 4 5.00 Very Low Very Low Very Low
Substantia nigra 3 5.00 Very Low Very Low Very Low
tetrodotoxin 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Convulsion 30 50.00 Quite Low
Stress 2 31.40 Quite Low
Epilepsy 22 5.00 Very Low Very Low Very Low
Status Epilepticus 3 5.00 Very Low Very Low Very Low
Hypoxia 2 5.00 Very Low Very Low Very Low
Injury 2 5.00 Very Low Very Low Very Low
Neurological Disease 1 5.00 Very Low Very Low Very Low
Hepatic Encephalopathy 1 5.00 Very Low Very Low Very Low
Syndrome 1 5.00 Very Low Very Low Very Low
Targeted Disruption 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In contrast, decrease in intracellular chloride occurs when potassium chloride cotransporters (KCCs), sodium dependent anion exchangers (NDAE) or chloride channel 2 (Clc2) are overexpressed.
Positive_regulation (overexpressed) of Gene_expression (overexpressed) of Clc2
1) Confidence 0.01 Published 2008 Journal Current Neuropharmacology Section Body Doc Link PMC2645547 Disease Relevance 0 Pain Relevance 0.06

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox