INT261787

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Context Info
Confidence 0.02
First Reported 2008
Last Reported 2008
Negated 0
Speculated 1
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.77
Pain Relevance 0.20

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

pigmentation (Fig4) extracellular region (Bdnf) Golgi apparatus (Fig4)
endoplasmic reticulum (Fig4) cytoplasmic membrane-bounded vesicle (Bdnf)
Bdnf (Rattus norvegicus)
Fig4 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
bDMF 10 100.00 Very High Very High Very High
Hippocampus 11 98.48 Very High Very High Very High
antagonist 4 9.88 Low Low
Glutamate receptor 2 9.40 Low Low
gABA 36 5.00 Very Low Very Low Very Low
agonist 6 5.00 Very Low Very Low Very Low
anticonvulsant 5 5.00 Very Low Very Low Very Low
GABAergic 5 5.00 Very Low Very Low Very Low
Calcium channel 4 5.00 Very Low Very Low Very Low
Substantia nigra 3 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Convulsion 30 96.64 Very High Very High Very High
Epilepsy 22 81.68 Quite High
Hypoxia 2 29.84 Quite Low
Malformations Of Cortical Development 1 29.36 Quite Low
Injury 2 28.16 Quite Low
Status Epilepticus 3 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low
Neurological Disease 1 5.00 Very Low Very Low Very Low
Hepatic Encephalopathy 1 5.00 Very Low Very Low Very Low
Syndrome 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
A possible explanation is that activation of BDNF and GABAAergic signaling during neonatal SE may actually upregulate KCC2 (Fig. 2).
Positive_regulation (activation) of BDNF Spec (may) Positive_regulation (upregulate) of Fig associated with bdmf
1) Confidence 0.02 Published 2008 Journal Current Neuropharmacology Section Body Doc Link PMC2645547 Disease Relevance 0.77 Pain Relevance 0.20

General Comments

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